Saccharomyces boulardii Forte (DBVPG 6763 strain/20 billion CFU/g) supports healthy microflora balance & gut health.*
This is a genetically and functionally distinct product from the typical saccharomyces cerevisiae sp. that is, in fact, brewer’s or baker’s yeast made in combination with lactose. Brewer’s yeast was very popular years ago for various conditions. However, the increase in allergies and sensitivities makes it no longer a viable nutritional remedy, particularly for those with learning disability challenges.
Saccharomyces boulardii Forte DBVPG 6763 is a unique strain of this significant freeze-dried live probiotic that requires no refrigeration to yield an exceptionally high CFU content. Strain verified through genomic typing to ensure maximum value.
S. boulardii is a viable yeast and, therefore, does not contain any milk products. S. boulardii is a true “biotic” or living organism that has been shown to have multiple beneficial effects via improving intestinal microbalance when orally administered. S. boulardii is not part of the naturally occurring gut flora and is not significantly impacted by antibiotics. In fact, S. boulardii has been shown to be supportive of antibiotic therapy.
In clinical studies, S. boulardii has been demonstrated to increase the production of secretory IgA (sIGA), the first-line defense immunoglobin of the gastrointestinal tract, crucial for supporting immune health, particularly in the gastrointestinal system and in mucosal secretions throughout the body, which are exposed to pathogenic organisms and viruses. When sIGA levels are depleted, a low immune response follows, often in the form of food intolerances, sensitivities, and digestive challenges such as dysbiosis. This opens the door wide for potentially harmful bacteria, Candida, yeasts, and parasites.
S. boulardii has been shown to inactivate bacterial toxins, stimulate intestinal immune response, and release polyamines, which are essential for normal cell growth and differentiation. These factors may help protect against enteric pathogens, improve tight junction structure, and, in dendritic immune cells, increase IL-10, which limits unspecific immune responses by down-regulating the Th1 response.